Int. J. Mol. Sci., Volume 21, Issue 21 (November-1 2020) – 607 articles
Cover Story (view full-size image): CaMKII and neuronal nitric oxide synthase (nNOS) co-localize in neurons, where CaMKII phosphorylates and inhibits nNOS via a specific Ser residue in transient forebrain ischemia, especially in hypothermic conditions. Meanwhile, CaMKII activities are reversibly regulated by oxidative environmental stress via its specific Cys residue. Thus, mutual regulation between CaMKII and NO might be potential therapeutic signals for neuronal injury. View this paper
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