Adaptive Evolution of Bacterial Pathogens in Response to Antibiotic Therapy
A special issue of Antibiotics (ISSN 2079-6382).
Deadline for manuscript submissions: closed (31 October 2021) | Viewed by 2707
Special Issue Editor
Special Issue Information
Dear Colleagues,
Bacterial pathogens have developed numerous methods to circumvent antibiotic therapy and cause long-term infections. In many instances, adaptations that occur during infection are pleiotropic and affect physiology, host-immune interactions, and pathogenesis.
Next-generation genome sequencing provides a technical platform to map and characterize adaptive mutations that are selected for during infection. The current Special Issue focuses on new mechanisms that alter treatment efficacy, immune responses, and/or bacterial virulence for the common nosocomial ‘ESKAPE’ pathogens (Enterococcus faecium, Staphylococcus aureus, Klebsiella pneumoniae, Acinetobacter baumannii, Pseudomonas aeruginosa, and Enterobacter spp), as well as for less-typical pathogens that may infrequently cause long-term human disease.
Dr. David Robert Cameron
Guest Editor
Manuscript Submission Information
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Submitted manuscripts should not have been published previously, nor be under consideration for publication elsewhere (except conference proceedings papers). All manuscripts are thoroughly refereed through a single-blind peer-review process. A guide for authors and other relevant information for submission of manuscripts is available on the Instructions for Authors page. Antibiotics is an international peer-reviewed open access monthly journal published by MDPI.
Please visit the Instructions for Authors page before submitting a manuscript. The Article Processing Charge (APC) for publication in this open access journal is 2900 CHF (Swiss Francs). Submitted papers should be well formatted and use good English. Authors may use MDPI's English editing service prior to publication or during author revisions.
Keywords
- Comparative genomics
- functional genomics
- microbial evolution
- antibiotic resistance
- pathogenesis
