Crosstalk between Epigenetic and Metabolic Mechanisms in Chronic Inflammation
A special issue of International Journal of Molecular Sciences (ISSN 1422-0067). This special issue belongs to the section "Molecular Immunology".
Deadline for manuscript submissions: closed (30 June 2022) | Viewed by 4179
Special Issue Editor
Interests: macrophages differentiation; macrophage metabolism and epigenetics; molecular mechanisms of chronic inflammation in cancer, diabetes, vascular complications, and tissue regeneration; macrophage biomarkers; biomaterials for macrophage drug delivery
Special Issues, Collections and Topics in MDPI journals
Special Issue Information
Dear colleagues,
Chronic inflammation is the cause for most life-threatening diseases that include cancer, diabetes and its vascular compilations, and neurodegeneration. Chronic inflammation, both systemic and local, can also result in delay in wound healing and implant failure. Epigenetic and metabolic mechanism are both critical for the detrimental programming of immune cells.
Our issue is dedicated to elucidation of the essential epigenetic and metabolic mechanism that controls inflammatory programming of differed cell types, and to identification of emerging hubs where the epigenetic and metabolic pathways interact with each other. Therefore, we invite submissions that cover interaction between glycolysis, fatty acid oxidation, oxidative protein phosphorylation, amino-acid metabolism, hypoxia and oxidative stress, genomic and mitochondrial DNA methylation, histone code, and miRNA. We aim to identify the promising pathways and critical molecules for therapeutic targeting to revert detrimental inflammatory programs in various cell types and restore homeostatic balance in tissue and organs.
Prof. Dr. Julia Kzhyshkowska
Guest Editor
Manuscript Submission Information
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Keywords
- diabetes
- atherosclerosis
- cancer
- macrophages
- T-cells
- endothelial cells
- epigenetic
- mitochondria
