Special Issue "Biological Role of Oxidative Stress in Inflammatory Diseases"
Deadline for manuscript submissions: 15 July 2021.
Interests: nutrition; cellular biology; oxidative stress; cardiovascular disease; endogenous antioxidant enzymes; bioactive vegetable; molecules
Interests: oxidative stress, nutrition; cellular biology; disease; antioxidant; bioactive vegetable; molecules; endogenous antioxidant enzymes; vegetable food; nitric oxide; inflammation and cardiovascular disease
Special Issues and Collections in MDPI journals
Special Issue in International Journal of Molecular Sciences: Oxidative Stress in Cardiovascular Disease 2018
Special Issue in Cells: Oxidative Stress, Nutrition and Cardiovascular Diseases
Assistant Guest Editor
Interests: inflammation; gel electrophoresis; agarose gel electrophoresis; neurodegeneration; neurodegenerative diseases
The imbalance between reactive oxidant species and endogenous antioxidant defense mechanisms promotes the development of a condition of oxidative stress, which has considerable biological consequences. Increasing evidence is proving that oxidative stress plays a pivotal role in the development and progression of inflammation, and thus contributes to the pathophysiology of a number of diseases, such as cancer, cardiovascular diseases, diabetes, and neurodegenerative processes.
Oxidant species influence all phases of the inflammatory response, including the release by damaged tissues of molecules acting as endogenous danger signals. Their sensing by innate immune receptors from the Toll-like (TLRs) and the NOD-like (NLRs) families leads to activation of signaling pathways promoting the adaptive cellular response to such signals.
While we tend to describe oxidative stress simply as harmful for the human body, it is also true that it is exploited as a therapeutic approach to treat clinical conditions such as cancer, with a certain degree of clinical success.
We can conclude that oxidative stress, although being one of the major injuries to individuals' wellness and health, can also be exploited as a treatment tool.
Therefore, correlations between oxidative stress, inflammation, and disease should be carefully investigated in order to better understand the etiopathogenesis of diseases and to develop ad hoc future treatments.
Prof. Dr. Sara Franceschelli
Prof. Lorenza Speranza
Dr. Chiara D’Angelo
Assistant Guest Editor
Manuscript Submission Information
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