Special Issue "TNF and ROS Crosstalk in Inflammation"
Deadline for manuscript submissions: 30 April 2021.
Special Issues and Collections in MDPI journals
Tumor necrosis factor-α (TNF-α) is a representative pro-inflammatory cytokine that contributes to host defense by inducing a wide variety of cellular responses, including cell growth, differentiation, inflammatory responses, and cell death. TNF-α also promotes production of reactive oxygen species (ROS) as second messengers to stimulate ROS-responsive signaling pathways. Conversely, previous studies demonstrated that signalling mechanisms activated by ROS upregulates the expression of TNF-α. Accumulating evidence indicates the involvement of intricate crosstalk between TNF-α and ROS in the pathological process of inflammatory diseases and cancer. Therefore, uncovering the molecular mechanisms of the crosstalk between TNF-α and ROS would lead to a better understanding of the pathogenetic mechanisms of TNF-α- and ROS-related diseases, which may be one of the strategies to overcome these diseases. In this Special Issue, studies of novel signalling mechanisms and pathological process mediated by TNF-α and ROS are welcome.
Dr. Takuya Noguchi
Manuscript Submission Information
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