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Nutrients
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Chronic Stress and Low-Grade Chronic Inflammation Impact on Body Composition...
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Chronic Stress and Low-Grade Chronic Inflammation Impact on Body Composition and the Role of Nutritional Mediation

A special issue of Nutrients (ISSN 2072-6643). This special issue belongs to the section "Nutrition and Metabolism".

Deadline for manuscript submissions: closed (1 February 2020) | Viewed by 7195

Special Issue Editor

Prof. Dr. Jasminka Ilich-Ernst

E-Mail Website
Guest Editor
Institute for Successful Longevity, Florida State University, Tallahassee, FL 32306, USA
Interests: nutrition; bone and body composition; osteosarcopenic adiposity syndrome; nutrition in chronic stress and low-grade chronic inflammation; precision nutrition in health and disease
Special Issues, Collections and Topics in MDPI journals

Special Issue Information

Dear Colleagues,

Chronic stress and low-grade chronic inflammation (LGCI) are key underlying causes of many diseases, including bone and body composition impairments. Osteosarcopenic obesity (OSO), a newly identified syndrome, signifies the simultaneous impairment of bone (osteopenia/osteoporosis), muscle (sarcopenia), and adipose tissue (as overt obesity, ectopic fat in visceral area, or infiltrated fat into bone and muscle). OSO has been associated with poor diet and various metabolic derangements. Hyperactivation of the hypothalamic–pituitary–adrenal (HPA) axis (caused by stress) propagates LGCI and disrupts the metabolism of bone, muscle, and adipose tissue, worsening OSO and/or any of its components.

This Special Issue of Nutrients entitled “Chronic stress and low-grade chronic inflammation impact on body composition and the role of nutritional mediation” welcomes the submission of both original research articles and reviews of the scientific literature. Manuscripts should address the mechanisms of OSO and/or the changes in bone lean and/or adipose tissue (or OSO) influenced by chronic stress or inflammation and possible nutritional mediations.

Prof. Dr. Jasminka Ilich-Ernst
Guest Editor

Manuscript Submission Information

Manuscripts should be submitted online at www.mdpi.com by registering and logging in to this website. Once you are registered, click here to go to the submission form. Manuscripts can be submitted until the deadline. All submissions that pass pre-check are peer-reviewed. Accepted papers will be published continuously in the journal (as soon as accepted) and will be listed together on the special issue website. Research articles, review articles as well as short communications are invited. For planned papers, a title and short abstract (about 100 words) can be sent to the Editorial Office for announcement on this website.

Submitted manuscripts should not have been published previously, nor be under consideration for publication elsewhere (except conference proceedings papers). All manuscripts are thoroughly refereed through a single-blind peer-review process. A guide for authors and other relevant information for submission of manuscripts is available on the Instructions for Authors page. Nutrients is an international peer-reviewed open access semimonthly journal published by MDPI.

Please visit the Instructions for Authors page before submitting a manuscript. The Article Processing Charge (APC) for publication in this open access journal is 2900 CHF (Swiss Francs). Submitted papers should be well formatted and use good English. Authors may use MDPI's English editing service prior to publication or during author revisions.

Keywords

  • Chronic stress
  • Low-grade chronic inflammation
  • Osteosarcopenic obesity and its components (osteopenia/osteoporosis, sarcopenia, obesity)
  • Nutritional impact (either bad or good)
  • Macro- and micronutrients
  • Bioactive compounds in food
  • Energy, alcohol, and caffeine consumption
  • Antioxidants
  • Western-type diet

Published Papers (1 paper)

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Review

20 pages, 2688 KiB  
Review
Chronic Stress Contributes to Osteosarcopenic Adiposity via Inflammation and Immune Modulation: The Case for More Precise Nutritional Investigation
by Jasminka Z. Ilich, Jennifer C. Gilman, Selma Cvijetic and Dario Boschiero
Nutrients 2020, 12(4), 989; https://0-doi-org.brum.beds.ac.uk/10.3390/nu12040989 - 02 Apr 2020
Cited by 26 | Viewed by 6747
Abstract
Chronic stress and low-grade chronic inflammation (LGCI) are key underlying factors for many diseases, including bone and body composition impairments. Objectives of this narrative review were to examine the mechanisms by which chronic stress and LGCI may influence osteosarcopenic adiposity (OSA) syndrome, originally [...] Read more.
Chronic stress and low-grade chronic inflammation (LGCI) are key underlying factors for many diseases, including bone and body composition impairments. Objectives of this narrative review were to examine the mechanisms by which chronic stress and LGCI may influence osteosarcopenic adiposity (OSA) syndrome, originally named as ostoesarcopenic obesity (OSO). We also examined the crucial nutrients presumed to be affected by or cause of stress and inflammation and compared/contrasted them to those of our prehistoric ancestors. The evidence shows that stress (particularly chronic) and its related inflammatory processes, contribute to osteoporosis, sarcopenia, and adiposity ultimately leading to OSA as a final and most deranged state of body composition, commencing at the mesenchymal cell lineage disturbance. The foods/nutrients consumed by modern humans, as well as their altered lifestyle, also contribute to stress, LGCI and subsequently to OSA. The processes can also go in opposite direction when stress and inflammation impact nutritional status, particularly some micronutrients’ levels. While nutritional management of body composition and LGCI have been studied, the nutrients (and their quantities) most affected by stressors and those which may act toward the alleviation of stressful state, ultimately leading to better body composition outcomes, need to be elucidated. Full article
(This article belongs to the Special Issue Chronic Stress and Low-Grade Chronic Inflammation Impact on Body Composition and the Role of Nutritional Mediation)
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