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Cells
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Exclusive Review Papers in "Cell Motility and Adhesion"
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Exclusive Review Papers in "Cell Motility and Adhesion"

A special issue of Cells (ISSN 2073-4409). This special issue belongs to the section "Cell Motility and Adhesion".

Deadline for manuscript submissions: closed (31 January 2024) | Viewed by 4904

Special Issue Editor

Prof. Nicoletta Gagliano

E-Mail Website
Guest Editor
Department of Biomedical Sciences for Health, Università degli Studi di Milano, 20133 Milan, Italy
Interests: collagen turnover; extracellular matrix remodeling; epithelial-to-mesenchymal transition; fibrosis; tendon biology
Special Issues, Collections and Topics in MDPI journals

Special Issue Information

Dear Colleagues,

Under the section “Cell Motility and Adhesion”, we aim to set up a Special Issue to publish high-quality review papers related to cell motility and adhesion. The topics of interest include, but are not limited to, the following:

  • Cytoskeleton;
  • Membrane trafficking;
  • Cellular dynamics;
  • Cell adhesion and cell junctions;
  • Cell division;
  • Cell migration;
  • Biotechnologies related to cell motility and adhesion;
  • Other topics fitting the section scope.

This Special Issue is intended to contribute to the compilation of the current knowledge on cell motility and adhesion, and to make it available to a broad readership. Distinguished researchers from all over the world, working in all related fields, are therefore invited to contribute to this review series. All papers will be published on an ongoing basis with full open access. We are looking forward to receiving your interesting contributions.

Prof. Dr. Nicoletta Gagliano
Guest Editor

Manuscript Submission Information

Manuscripts should be submitted online at www.mdpi.com by registering and logging in to this website. Once you are registered, click here to go to the submission form. Manuscripts can be submitted until the deadline. All submissions that pass pre-check are peer-reviewed. Accepted papers will be published continuously in the journal (as soon as accepted) and will be listed together on the special issue website. Research articles, review articles as well as short communications are invited. For planned papers, a title and short abstract (about 100 words) can be sent to the Editorial Office for announcement on this website.

Submitted manuscripts should not have been published previously, nor be under consideration for publication elsewhere (except conference proceedings papers). All manuscripts are thoroughly refereed through a single-blind peer-review process. A guide for authors and other relevant information for submission of manuscripts is available on the Instructions for Authors page. Cells is an international peer-reviewed open access semimonthly journal published by MDPI.

Please visit the Instructions for Authors page before submitting a manuscript. The Article Processing Charge (APC) for publication in this open access journal is 2700 CHF (Swiss Francs). Submitted papers should be well formatted and use good English. Authors may use MDPI's English editing service prior to publication or during author revisions.

Published Papers (2 papers)

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Review

16 pages, 1071 KiB  
Review
Focal Adhesion’s Role in Cardiomyocytes Function: From Cardiomyogenesis to Mechanotransduction
by Simona Casarella, Federica Ferla, Dalila Di Francesco, Elena Canciani, Manuela Rizzi and Francesca Boccafoschi
Cells 2024, 13(8), 664; https://0-doi-org.brum.beds.ac.uk/10.3390/cells13080664 - 10 Apr 2024
Viewed by 932
Abstract
Mechanotransduction refers to the ability of cells to sense mechanical stimuli and convert them into biochemical signals. In this context, the key players are focal adhesions (FAs): multiprotein complexes that link intracellular actin bundles and the extracellular matrix (ECM). FAs are involved in [...] Read more.
Mechanotransduction refers to the ability of cells to sense mechanical stimuli and convert them into biochemical signals. In this context, the key players are focal adhesions (FAs): multiprotein complexes that link intracellular actin bundles and the extracellular matrix (ECM). FAs are involved in cellular adhesion, growth, differentiation, gene expression, migration, communication, force transmission, and contractility. Focal adhesion signaling molecules, including Focal Adhesion Kinase (FAK), integrins, vinculin, and paxillin, also play pivotal roles in cardiomyogenesis, impacting cell proliferation and heart tube looping. In fact, cardiomyocytes sense ECM stiffness through integrins, modulating signaling pathways like PI3K/AKT and Wnt/β-catenin. Moreover, FAK/Src complex activation mediates cardiac hypertrophic growth and survival signaling in response to mechanical loads. This review provides an overview of the molecular and mechanical mechanisms underlying the crosstalk between FAs and cardiac differentiation, as well as the role of FA-mediated mechanotransduction in guiding cardiac muscle responses to mechanical stimuli. Full article
(This article belongs to the Special Issue Exclusive Review Papers in "Cell Motility and Adhesion")
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15 pages, 1263 KiB  
Review
LFA-1 Activation in T-Cell Migration and Immunological Synapse Formation
by Huiping Shi and Bojing Shao
Cells 2023, 12(8), 1136; https://0-doi-org.brum.beds.ac.uk/10.3390/cells12081136 - 12 Apr 2023
Cited by 2 | Viewed by 3600
Abstract
Integrin LFA-1 plays a critical role in T-cell migration and in the formation of immunological synapses. LFA-1 functions through interacting with its ligands with differing affinities: low, intermediate, and high. Most prior research has studied how LFA-1 in the high-affinity state regulates the [...] Read more.
Integrin LFA-1 plays a critical role in T-cell migration and in the formation of immunological synapses. LFA-1 functions through interacting with its ligands with differing affinities: low, intermediate, and high. Most prior research has studied how LFA-1 in the high-affinity state regulates the trafficking and functions of T cells. LFA-1 is also presented in the intermediate-affinity state on T cells, however, the signaling to activate LFA-1 to the intermediate-affinity state and the role of LFA-1 in this affinity state both remain largely elusive. This review briefly summarizes the activation and roles of LFA-1 with varied ligand-binding affinities in the regulation of T-cell migration and immunological synapse formation. Full article
(This article belongs to the Special Issue Exclusive Review Papers in "Cell Motility and Adhesion")
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